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The -subunit of the K28 toxin precursor contains a classical ER-retention signal (HDEL) that is initially masked by a C-terminal arginine residue, allowing the toxin precursor to pass through the secretory pathway from the ER to the Golgi

Although LOX and LOXLs are reported to significantly promote fibrotic alterations in tissues, certain stimuli are required to stimulate fibroblasts or myofibroblasts to secrete their precursors (91,92)

(2019) Role of GPX4 in ferroptosis and its pharmacological implication, Free Radic