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Ethionamide requires activation by the mycobacterial enzyme flavin monooxygenase (EthA) and the transcriptional repressor EthR to form ethionamide sulfoxide, which binds to NAD + and inhibits the enoyl-acyl carrier protein reductase (InhA), thereby blocking mycolic acid synthesis, responsible for cell death (Dover et al., 2007)

References: National Institute of Health (NIH): Smoking Wet NIH: Phencyclidine Intoxication and Adverse Effects: A Clinical and Pharmacological Review of an Illicit Drug NIH: The Use of Fry (Embaling Fluid and PCP-laced Ciragettes or Marijuana Sticks) Among Crack Cocaine Users Science Direct: Phencyclidine (PCP): Some human studies Jump to a Section Call (855) 425-4846